The risk for developing the sporadic late-onset form of Alzheimer's disease is attributable to an interaction of genetic and environmental factors. Among the genetic factors, it is well known that the presence of the APOE4 gene considerably increases the risk of developing the disease. Among environmental risk factors, lifestyle measures (such as diet, sleep quality, physical activity, and mental training) and exposure to toxic agents have great importance for prevention, since they can be modified both at the individual level and through collective health policies. Despite its relevance, the effect of exposure to toxins has not been properly studied: the number of articles published on this subject is limited, and occur mostly in journals on environment and toxins rather than in medical journals.
The coconut palm (cocos nucifera), also known as the "tree of life," provides many valuable foods - most famously coconut oil. In recent years, global consumption of coconut oil has boomed due to its promised health benefits. But does the consumption of coconut oil really bring health benefits or does it rather do harm?
There is a diet that for many years has already achieved the best results in the fight against the common ailments of civilization: the LOGI method. Whether in the prevention or treatment of metabolic syndrome, type 2 diabetes, lipid metabolism disorders such as high triglycerides and low HDL cholesterol, whether for the follow-up of non-alcoholic fatty liver, against high blood pressure, polycystic ovarian syndrome or simply "just" to stay in shape - with the help of the LOGI method it is all possible.
Since 2022 the team of the project "Knowledge stops Dementia" started to refer on a regular base to current contributions from the world wide web which have a close relation to our core topic, the early detection of individual risk factors and thus the lifestyle-oriented and self-responsible prevention and treatment of dementia-related diseases. The opinions expressed in the following articles should serve as a stimulus for critical discussion. They do not necessarily correspond to the positions we hold, but in all cases, they enrich the basis for discussion. Occasionally, we also refer to current freely available studies at the end – please make up your own mind!
Spermidine, also known chemically as N-(3-aminopropyl) butane-1,4-diamine, is one of the naturally occurring polyamines. It occurs as a natural substance in amino acid metabolism of all living organisms and in all cells, and therefore also in a variety of plant and animal foods. Particularly rich food sources are wheat germ, but hard cheese, dried soybeans and some other foods also provide good amounts of spermidine. Recently, it was shown in mice that spermidine from food actually reaches the brain, thus crossing the blood-brain barrier.
Can cataract surgery lower the risk of dementia? – a new study shows how good vision in elderly can protect cognition.
The primary function of the nervous system is to adapt the living being to the environment. In order to accomplish [...]
Vitamin E supplements and Alzheimer’s disease: a new study supports the importance of its composition.
The use of vitamin E supplements in the prevention and treatment of Alzheimer’s disease has been studied for a long time and still controversial: The scientific [...]
With the beginning of the New Year, the team of the project "Knowledge stopps Dementia" would like to refer on a regular base to current contributions from the world wide web which have a close relation to our core topic, the lifestyle-oriented and self-responsible prevention and treatment of dementia-related diseases. The opinions expressed in the following articles should serve as a stimulus for critical discussion. They do not necessarily correspond to the positions we hold, but in all cases, they enrich the basis for discussion. Occasionally, we also refer to current freely available studies at the end - make up your own mind!
During my medical residency in neurology at the Hospital of Federal Fluminense University (UFF) in Rio de Janeiro/Brazil we often faced the following question: some medications commonly prescribed in routine neurology practice (for patients with Parkinson's syndrome, depression or urinary alterations) inhibited the neurotransmitter acetylcholine, important for the formation and maintenance of memory. Since one of the proposals for the pharmacological treatment of Alzheimer's disease is precisely to increase the concentration of acetylcholine in the brain, the use of drugs that inhibit this neurotransmitter seemed contradictory and threatening to us.