There is a diet that for many years has already achieved the best results in the fight against the common ailments of civilization: the LOGI method. Whether in the prevention or treatment of metabolic syndrome, type 2 diabetes, lipid metabolism disorders such as high triglycerides and low HDL cholesterol, whether for the follow-up of non-alcoholic fatty liver, against high blood pressure, polycystic ovarian syndrome or simply "just" to stay in shape - with the help of the LOGI method it is all possible.
Since 2022 the team of the project "Knowledge stops Dementia" started to refer on a regular base to current contributions from the world wide web which have a close relation to our core topic, the early detection of individual risk factors and thus the lifestyle-oriented and self-responsible prevention and treatment of dementia-related diseases. The opinions expressed in the following articles should serve as a stimulus for critical discussion. They do not necessarily correspond to the positions we hold, but in all cases, they enrich the basis for discussion. Occasionally, we also refer to current freely available studies at the end – please make up your own mind!
Spermidine, also known chemically as N-(3-aminopropyl) butane-1,4-diamine, is one of the naturally occurring polyamines. It occurs as a natural substance in amino acid metabolism of all living organisms and in all cells, and therefore also in a variety of plant and animal foods. Particularly rich food sources are wheat germ, but hard cheese, dried soybeans and some other foods also provide good amounts of spermidine. Recently, it was shown in mice that spermidine from food actually reaches the brain, thus crossing the blood-brain barrier.
Can cataract surgery lower the risk of dementia? – a new study shows how good vision in elderly can protect cognition.
The primary function of the nervous system is to adapt the living being to the environment. In order to accomplish [...]
Vitamin E supplements and Alzheimer’s disease: a new study supports the importance of its composition.
The use of vitamin E supplements in the prevention and treatment of Alzheimer’s disease has been studied for a long time and still controversial: The scientific [...]
With the beginning of the New Year, the team of the project "Knowledge stopps Dementia" would like to refer on a regular base to current contributions from the world wide web which have a close relation to our core topic, the lifestyle-oriented and self-responsible prevention and treatment of dementia-related diseases. The opinions expressed in the following articles should serve as a stimulus for critical discussion. They do not necessarily correspond to the positions we hold, but in all cases, they enrich the basis for discussion. Occasionally, we also refer to current freely available studies at the end - make up your own mind!
During my medical residency in neurology at the Hospital of Federal Fluminense University (UFF) in Rio de Janeiro/Brazil we often faced the following question: some medications commonly prescribed in routine neurology practice (for patients with Parkinson's syndrome, depression or urinary alterations) inhibited the neurotransmitter acetylcholine, important for the formation and maintenance of memory. Since one of the proposals for the pharmacological treatment of Alzheimer's disease is precisely to increase the concentration of acetylcholine in the brain, the use of drugs that inhibit this neurotransmitter seemed contradictory and threatening to us.
With the project "Knowledge stops Dementia", the German Foundation for Health Information and Prevention (Deutsche Stiftung für Gesundheitsinformation und Prävention DSGiP), together with the Academy for Human Medicine (Akademie für menschliche Medizin), presents a broad spectrum of lifestyle-oriented measures for the prevention and therapy of Alzheimer's disease. In addition to Alzheimer's dementia, however, other neurodegenerative diseases such as multiple sclerosis and Parkinson's disease also pose a major challenge for those affected, their relatives and, ultimately, the healthcare system, since the prospect of a cure through pharmaceutical approaches is still a long way in the future. But the good news is, individualized lifestyle concepts seem to be effective also in these diseases.
Our brain is one of the largest glucose consumers in our body. Not all areas of the brain need the hormone insulin to take up glucose and thus operate insulin-independently. However, this is different in the neurons of the hippocampus, the brain region responsible for our memory consolidation and one of the first to be affected by Alzheimer's and dementia: here - as researchers have recently discovered - insulin is needed to provide the neurons with sufficient glucose supply during times of increased energy demand. Thus, it's not surprising that when insulin resistance is present, for example during developing or established type-2 diabetes, the brain can also suffer from a glucose deficiency. This impaired glucose utilization and the associated energy crisis in the brain have also been visualized in imaging techniques, the so-called PET scan, in Alzheimer's patients.